Author

A. Eldar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Organism:bacteria (other)· 1

Frequent Co-Authors

N. Aframian· 3 shared P. Guler· 3 shared I. Grinberg· 2 shared S. Omer Bendori· 2 shared A. D. Grossman· 1 shared A. Kessel· 1 shared A. Mahagna· 1 shared A. Stokar-Avihail· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersMicrobiology

N. Aframian

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

High defense system expression broadens protection range at the cost of increased autoimmunity

N. Aframian, S. Omer-Bendori, T. Hen, P. Guler, A. Eldar

This study shows that increasing defense system expression in bacteria broadens protection against phages but increases autoimmunity, revealing a tradeoff in defense system regulation.

bioRxiv · Nov 30, 2023 · PreprintMicrobiology

Arbitrium communication controls phage life-cycle through modulation of a bacterial anti-phage defense system

P. Guler, S. Omer Bendori, N. Aframian, A. Kessel, A. Eldar

This study shows how phage phi3T uses the arbitrium system to modulate the MazEF toxin-antitoxin system, controlling the lysis/lysogeny decision in bacteria.

bioRxiv · Apr 27, 2023 · PreprintMicrobiology

Dormant phages communicate to control exit from lysogeny

N. Aframian, S. Omer Bendori, S. Hen, P. Guler, A. Stokar-Avihail, E. Manor, k. Msaeed, V. Lipsman, I. Grinberg, A. Mahagna, A. Eldar

This study shows that phages use the arbitrium signaling system to regulate both entry into and exit from lysogeny, reducing induction rates when surrounded by other lysogens.

bioRxiv · Sep 20, 2021 · PreprintMicrobiology

A mobile genetic element increases bacterial host fitness by manipulating development

J. M. Jones, I. Grinberg, A. Eldar, A. D. Grossman

This study shows that the mobile genetic element ICEBs1 enhances bacterial host fitness by manipulating development during biofilm formation and sporulation.

bioRxiv · Oct 9, 2020Microbiology