Author

A. Ghodousi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:whole-genome sequencing· 2

Frequent Co-Authors

D. M. Cirillo· 2 shared A. Ghafoor· 1 shared A. H. Rizvi· 1 shared A. Q. Baloch· 1 shared A. Trovato· 1 shared C. U. Köser· 1 shared E. Borroni· 1 shared E. Karunaratne· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersMicrobiology

D. M. Cirillo

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Acquisition of cross-resistance to bedaquiline and clofazimine following treatment for tuberculosis in Pakistan

A. Ghodousi, A. H. Rizvi, A. Q. Baloch, A. Ghafoor, F. M. Khanzada, M. Qadir, E. Borroni, A. Trovato, S. Tahseen, D. M. Cirillo

The paper reports six cases of acquired resistance to bedaquiline and clofazimine in Pakistan, documenting mutations and MIC increases in tuberculosis patients on bedaquiline-containing regimens.

bioRxiv · May 7, 2019Microbiology

Isoniazid resistance in Mycobacterium tuberculosis is a heterogeneous phenotype comprised of overlapping MIC distributions with different underlying resistance mechanisms

A. Ghodousi, E. Tagliani, E. Karunaratne, S. Niemann, J. Perera, C. U. Köser, D. M. Cirillo

The paper analyzes isoniazid resistance in M. tuberculosis strains, finding overlapping MIC distributions and showing that whole-genome sequencing can explain most resistance levels.

bioRxiv · Jan 20, 2019Microbiology