Author

D. Ghersi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 4 Research Focus:cell type annotation· 3 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Gil· 3 shared L. K. Selin· 3 shared N. Aslan· 3 shared K. Luzuriaga· 2 shared L. Kamga· 2 shared A. K. Ganti· 1 shared F. Clark· 1 shared F. G. Clark· 1 shared

Activity by Year

2018-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersImmunology

A. Gil

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Co-evolution of human influenza A and Epstein Barr virus-specific CD8 ex vivo memory T cell receptor BV repertoires with increasing age

F. Clark, A. Gil, N. Aslan, D. Ghersi, L. K. Selin

This study shows that CD8 memory T cell receptor BV repertoires for influenza A and Epstein-Barr virus co-evolve with age, narrowing in diversity and focusing on common TRBV in older individuals.

bioRxiv · Dec 15, 2022 · PreprintImmunology

1q21.3 amplification modulates therapy sensitivity in Triple-Negative Breast Cancer

R. Chirravuri Venkata, D. Ghersi, A. K. Ganti, I. Lakshmanan, S. Chaudhary, S. K. Batra, M. W. Nasser

The paper identifies 1q21.3 amplification as a genetic marker linked to therapy sensitivity in Triple-Negative Breast Cancer.

bioRxiv · Apr 29, 2020 · PreprintCancer Biology

Epstein Barr virus epitope/MHC interaction combined with convergent recombination drive selection of diverse T cell receptor a and b repertoires

A. Gil, L. Kamga, R. Chirravuri-Venkata, N. Aslan, F. G. Clark, D. Ghersi, K. Luzuriaga, L. K. Selin

The paper shows that convergent recombination and MHC epitope interactions drive diverse TCR a and b repertoires during EBV infection, with persistent clonotypes having distinct features.

bioRxiv · Feb 6, 2020Immunology

High diversity, turnover, and structural constraints characterize TCR α and β repertoire selection

L. Kamga, A. Gil, I. Song, R. Chirravuri, N. Aslan, D. Ghersi, L. J. Stern, L. K. Selin, K. Luzuriaga

The paper examines TCR α and β repertoire dynamics during EBV infection, finding that persistent clonotypes are highly expanded in acute infection and replaced by new clonotypes in convalescence.

bioRxiv · Sep 29, 2018 · PreprintImmunology