Author

E. Duran

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 3

Signature Assays, Organisms & Methods

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Assay:imaging· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

A. Gonzalo· 3 shared J. Bermejo· 3 shared J. C. del Alamo· 3 shared O. Flores· 3 shared P. Martinez-Legazpi· 3 shared E. McVeigh· 2 shared M. Garcia-Villalba· 2 shared A. Kahn· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersBioengineering

A. Gonzalo

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Author profile →

3 papers

Recent·Most cited

Multi-physics simulations reveal hemodynamic impacts of patient-derived fibrosis-related changes in left atrial tissue mechanics

A. Gonzalo, C. Augustin, S. F. Bifulco, A. Telle, Y. Chahine, A. Kassar, M. Guerrero-Hurtado, E. Duran, P. Martinez-Legazpi, O. Flores, J. Bermejo, G. Plank, N. Akoum, P. M. Boyle, J. C. del Alamo

This paper uses multi-physics simulations to show how fibrosis in left atrial tissue affects hemodynamics and increases stroke risk by altering flow patterns and reducing emptying fractions.

bioRxiv · May 29, 2024 · PreprintBioengineering

Pulmonary vein flow split effects in patient-specific simulations of left atrial flow

E. Duran, M. Garcia-Villalba, P. Martinez-Legazpi, A. Gonzalo, E. McVeigh, A. M. Kahn, J. Bermejo, O. Flores, J. C. del Alamo

This study examines how variations in pulmonary vein flow split affect left atrial flow and thrombosis risk using patient-specific simulations.

bioRxiv · Mar 24, 2023Bioengineering

Non-Newtonian Blood Rheology Impacts Left Atrial Stasis in Patient-Specific Simulations

A. Gonzalo, M. Garcia-Villalba, L. Rossini, E. Duran, D. Vigneault, P. Martinez-Legazpi, O. Flores, J. Bermejo, E. McVeigh, A. Kahn, J. C. del Alamo

This study shows that non-Newtonian blood rheology, influenced by hematocrit and rouleaux formation, increases left atrial stasis in patients with atrial fibrillation.

bioRxiv · Jun 24, 2021Bioengineering