Author

I. de Heer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

M. de Wit· 2 shared A. Brandes· 1 shared A. Gijtenbeek· 1 shared A. Nowak· 1 shared B. Baumert· 1 shared B. Valkenburg· 1 shared C. McBain· 1 shared D. Mercieca· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

M. de Wit

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintDec 10, 2020

DNA methylation and survival differences associated with the type of IDH mutation in 1p/19q non-codeleted astrocytomas

M. Tesileanu, W. Vallentgoed, M. Sanson…·van den Bent

The paper shows that IDH mutations other than p.R132H in 1p/19q non-codeleted astrocytomas are linked to higher DNA methylation and better patient survival.

DOI 10.1101/2020.12.10.419333

humanDNA methylation

Cancer BiologybioRxivPreprintOct 21, 2019

Effective Tyrosine Kinase Inhibitors result in the intracellular accumulation of EGFR and allows response prediction in patients

M. de Wit, Y. Gao, D. Mercieca…·P. French

The paper shows that EGFR tyrosine kinase inhibitors (TKIs) cause intracellular EGFR accumulation only in specific mutation-TKI combinations, which predicts clinical response in patients.

DOI 10.1101/798314

humanproteomics