Author

J. Dopazo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:whole-genome sequencing· 2 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:metabolomics· 1

Frequent Co-Authors

C. S. Casimiro-Soriguer· 2 shared F. Garcia· 2 shared J. A. Lepe· 2 shared J. Perez-Florido· 2 shared M. Lara· 2 shared N. Lorusso· 2 shared A. Aguado· 1 shared A. de Salazar· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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2 shared papersMicrobiology

C. S. Casimiro-Soriguer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SIEGA the integrated genomic surveillance system of Andalusia, a One Health regional resource connected with the clinic

C. S. Casimiro-Soriguer, J. Perez-Florido, E. A. Robles, M. Lara, A. Aguado, M. A. R. Iglesias, J. A. Lepe, F. Garcia, M. Perez-Alegre, E. Andujar, V. E. Jimenez, L. P. Camino, N. Lorusso, U. Ameyugo, I. M. Vazquez, , J. Dopazo

SIEGA is a genomic surveillance system that integrates data from humans, animals, and the environment to monitor infectious diseases and antimicrobial resistance.

bioRxiv · Feb 29, 2024 · PreprintMicrobiology

Molecular and phylogenetic characterization of the monkeypox outbreak in the South of Spain

C. S. Casimiro-Soriguer, J. Perez-Florido, M. Lara, P. Camacho-Martinez, L. Merino-Diaz, I. Pupo-Ledo, A. de Salazar, A. Fuentes, L. Vinuela, N. Chueca, L. Martinez-Martinez, N. Lorusso, J. A. Lepe, J. Dopazo, F. Garcia

The paper characterizes the monkeypox outbreak in southern Spain using whole genome sequencing and phylogenetic analysis, identifying mutations potentially linked to drug resistance.

bioRxiv · Sep 20, 2023Microbiology

Accelerated phosphatidylcholine turnover in macrophages promotes adipose tissue inflammation in obesity

K. Petkevicius, S. Virtue, G. Bidault, B. Jenkins, C. Cubuk, C. Morgantini, M. Aouadi, J. Dopazo, M. Serlie, A. Koulman, A. Vidal-Puig

This study shows that increased phosphatidylcholine synthesis in macrophages during obesity drives inflammation in adipose tissue, contributing to insulin resistance.

bioRxiv · May 8, 2019Cell Biology