Author

Rahul Roy

Northeastern UniversityPrimary ORCID profile (0000-0003-3329-8803 · 6 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Rahul RoyiD6 papers · Indian Institute of Science Bangalore

Biology Disciplines

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Bioinformatics· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:machine learning (classical)· 1 Research Focus:software / tool· 1 Approach:statistical model· 1

Frequent Co-Authors

Alok Dinesh Sharma· 1 shared Amitesh BadkuliD· 1 shared Amos Jaimes· 1 shared Anton V. SinitskiyiD· 1 shared Arjun Shrivatsan· 1 shared Arpita Kachhwah· 1 shared Casual Dineshbhai Kalotra· 1 shared Francesca FicaraiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperBioinformatics

Alok Dinesh Sharma

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2 papers

Recent·Most cited

Real Science Is Harder Than Benchmarks: Evaluating Advanced AI Frameworks on Published Studies. I. Uncertainty Quantification, ML on Therapeutic Data Commons, and Agent-Based Modeling

Mohammed O. Ahmed, Sahil Anil Amale, Rhythm Dipak Bhavsar, Pratham Chopra, Amos Jaimes, Arpita Kachhwah, Casual Dineshbhai Kalotra, Peizhao Li, Xingbei Li, Yuantian Liao, Rahul Roy, Nivethini Senthilselvan, Yukun Shao, Alok Dinesh Sharma, Arjun Shrivatsan, , Anton V. Sinitskiy

This paper evaluates five AI frameworks on real scientific tasks, finding they excel in hypothesis generation and reporting but struggle with depth and reliability in real-world research.

bioRxiv · Jun 27, 2026 · PreprintBioinformatics

A Unifying Mechanism for Shared Splicing Aberrations in Splicing Factor Mutant Cancers

Prajwal Chaitanya Boddu, Rahul Roy, Stephan Hütter, Francis Baumgartner, Wenxue Li, Gabriele Todisco, Francesca Ficara, Matteo Giovanni Della Porta, Yansheng Liu, Torsten Haferlach, Manoj M. Pillai

This study identifies a shared trans-acting mechanism in splicing factor mutant cancers that drives retained intron (RI) splicing defects through DDR-induced AMPK-AKT imbalance and SRSF1 hypophosphorylation.

bioRxiv · Oct 8, 2025 · PreprintCancer Biology