Astrocyte TrkB.T1 deficiency disrupts glutamatergic synaptogenesis and astrocyte-synapse interactions.
Beatriz T. C. Pinkston, Jack L. Browning, Amelie P. Larson, Michelle Lynne Olsen
This study shows that astrocyte TrkB.T1 is essential for synapse formation and astrocyte-synapse interactions, with loss of TrkB.T1 reducing synapse density and interactions.
Journal of Neuroscience · Jul 10, 2026Peer-reviewedNeuroscience