Author

Anna Karlik

Institute of Bioorganic Chemistry, Polish Academy of Sciences

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Abigael P. WilliamsiD· 1 shared Adriana GrabowskaiD· 1 shared Anna‐Maria BarciszewskaiD· 1 shared Dorota WronkaiD· 1 shared Erola Ainsua‐EnrichiD· 1 shared Harini BagavantiD· 1 shared Ibrahim HatipogluiD· 1 shared José Alberola‐IlaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

Abigael P. WilliamsORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

ANDROGENS PROTECT ILC2S FROM FUNCTIONAL SUPPRESSION DURING INFLUENZA VIRUS INFECTION

Sapana Kadel, Reegan Miller, Anna Karlik, Abigael P. Williams, Seán Turner, Erola Ainsua‐Enrich, Ibrahim Hatipoglu, Joshua W. Garton, Richard C. Pelikan, Harini Bagavant, José Alberola‐Ila, Susan Kovats

This study shows that androgens protect ILC2s from functional suppression during influenza virus infection by regulating IFNGR-STAT1 signaling.

Mucosal Immunology · Mar 27, 2026Peer-reviewedImmunology

Circular RNA - circCLIP2 is predominantly expressed in GSC niche and enhances glioblastoma aggressiveness via EMT and ECM signaling

Julia Oliwia Misiorek, Żaneta Zarębska, Konrad Kuczyński, Julia Latowska-Lysiak, Adriana Grabowska, Paweł Głodowicz, Marcin Piotr Sajek, Anna Karlik, Dorota Wronka, Anna‐Maria Barciszewska, Łukasz Przybył, Katarzyna Rolle

This study identifies circCLIP2 as a circRNA overexpressed in glioblastoma stem cells that promotes tumor aggressiveness through EMT and ECM signaling.

bioRxiv · Dec 1, 2024 · PreprintCancer Biology