Author

B. Amulic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. C. Williams· 1 shared A. K. Holt· 1 shared B. Hube· 1 shared C. F. Urban· 1 shared C. J. Bull· 1 shared D. N. Legge· 1 shared D. Qualtrough· 1 shared E. Backman· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. C. Williams

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Candida albicans promotes neutrophil extracellular trap formation and leukotoxic hypercitrullination via the peptide toxin candidalysin.

L. Unger, E. Backman, S. Yellagunda, F. M. Ponce-Garcia, B. Amulic, K. Renate, H. von Bernuth, J. Bylund, B. Hube, J. R. Naglik, C. F. Urban

This study shows candidalysin from C. albicans hyphae promotes both NETs and NLS, which together inhibit fungal growth and explain conflicting findings on ROS dependence in neutrophil responses.

bioRxiv · Oct 13, 2022 · PreprintImmunology

Identifying targetable metabolic dependencies across colorectal cancer progression

D. N. Legge, E. Stanko, A. K. Holt, C. J. Bull, T. J. Collard, M. Kollareddy, J. Bellamy, S. Groves, E. H. Ma, E. Hazelwood, D. Qualtrough, B. Amulic, K. N. Malik, A. C. Williams, N. Jones, E. E. Vincent

This study identifies asparagine synthesis as a metabolic vulnerability in late-stage colorectal cancer cells, showing that blocking asparagine synthetase inhibits their proliferation.

bioRxiv · Mar 23, 2022Cancer Biology