Author

C. Fellmann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

J. A. Doudna· 2 shared R. J. Lew· 2 shared A. M. Amen· 1 shared A. M. McKinney· 1 shared A. Mancini· 1 shared B. McMahon· 1 shared G. J. Knott· 1 shared H. Shivram· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

J. A. Doudna

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2 papers

Recent·Most cited

Cancer-specific loss of TERT activation sensitizes glioblastoma to DNA damage

A. M. Amen, C. Fellmann, K. M. Soczek, S. M. Ren, R. J. Lew, G. J. Knott, J. E. Park, A. M. McKinney, A. Mancini, J. A. Doudna, J. F. Costello

This study shows that reducing GABPB1L in TERT-mutant glioblastomas, combined with chemotherapy, reduces tumor growth by impairing DNA damage response.

Proceedings of the National Academy of Sciences · Mar 23, 2021Peer-reviewedCancer Biology

Potent CRISPR-Cas9 inhibitors from Staphylococcus genomes

K. E. Watters, H. Shivram, C. Fellmann, R. J. Lew, B. McMahon, J. A. Doudna

This study identifies three new potent CRISPR-Cas9 inhibitors from Staphylococcus genomes, showing they specifically block SauCas9 activity in human cells.

bioRxiv · Oct 9, 2019Biochemistry