Caleb CS Calhoun

The paper identifies dysregulation of cholesterol metabolism as a shared molecular consequence of schizophrenia risk mutations in zebrafish models.

This study removes 45 microexons in zebrafish using CRISPR/Cas9 and finds minimal developmental impact on brain morphology and function, with some exceptions in specific microexons.

This study identifies diencephalic and neuropeptidergic dysfunction in zebrafish with autism-risk mutations, revealing potential molecular and behavioral mechanisms.