Author

D. Anastsakis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:multiome· 1

Frequent Co-Authors

C. Hourigan· 1 shared D. Larson· 1 shared F. Liang· 1 shared K. Lindblad· 1 shared M. Hafner· 1 shared M. Palangat· 1 shared R. Bradley· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

C. Hourigan

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1 paper

Recent·Most cited

The splicing factor U2AF1 contributes to cancer progression through a non-canonical role in translation regulation

M. Palangat, D. Anastsakis, F. Liang, K. Lindblad, R. Bradley, C. Hourigan, M. Hafner, D. Larson

This study identifies a non-canonical role of U2AF1 in translation regulation, showing that the S34F mutation enhances interleukin 8 synthesis, promoting cancer progression.

bioRxiv · Jul 18, 2018Cell Biology