Author

David C. Quist

Fred Hutch Cancer Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

Abigail G. ParrishiD· 1 shared Alex Shimura YamashitaiD· 1 shared Betemariyam N. Gessesse· 1 shared Debra K. Kumasaka· 1 shared Dmytro RudoyiD· 1 shared Eric C. HollandiD· 1 shared Frank SzulzewskyiD· 1 shared James Z. Yan· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Abigail G. ParrishORCID

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1 paper

Recent·Most cited

ERK signaling promotes resistance to TRK kinase inhibition in NTRK fusion-driven glioma mouse models.

Sebastian Schmid, Zachary R. Russell, Alex Shimura Yamashita, Madeline E. West, Abigail G. Parrish, Julia Rose Walker, Dmytro Rudoy, James Z. Yan, David C. Quist, Betemariyam N. Gessesse, Neriah Alvinez, Kimberly D. Hill, Larry W. Anderson, Patrick Joseph Cimino, Debra K. Kumasaka, , Frank Szulzewsky

This study shows ERK signaling promotes resistance to TRK inhibitors in NTRK fusion-driven glioma mouse models and suggests MEK inhibition as a potential combination therapy.

Cell Reports · Oct 1, 2024Peer-reviewedCancer Biology