Author

David Mastro

University of California, Los Angeles

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Immunology· 1

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Alexander HoffmanniD· 1 shared Apeksha SinghiD· 1 shared Dennis J. ChiaiD· 1 shared Dinesh S. RaoiD· 1 shared Jennifer K. KingiD· 1 shared Noa PopkoiD· 1 shared Tiffany M. TraniD· 1 shared Yi Liu· 1 shared

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2025-2025 · peak 1/yr

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9 top collaborators · 36 mutual co-author links

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1 shared paperImmunology

Alexander HoffmannORCID

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1 paper

Recent·Most cited

Distinct causes of three phenotypic hallmarks of hematopoietic aging

Dennis J. Chia, Apeksha Singh, Yusheng Lin, Noa Popko, David Mastro, Yi Liu, Tiffany M. Tran, Jennifer K. King, Dinesh S. Rao, Alexander Hoffmann

This study identifies distinct causes for three hematopoietic aging phenotypes using mouse models and single-cell analysis, showing HSC accumulation is NFκB-independent, functional impairment is HSC-autonomous NFκB-driven, and myeloid bias is driven by the NFκB-altered bone marrow environment.

Cell Reports · Aug 26, 2025Peer-reviewedImmunology