Author

Eva Rodriguez

Hospital Del Mar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Organism:mouse· 1

Frequent Co-Authors

Adria Caballe· 1 shared Aishwarya Nene· 1 shared Alan GutierreziD· 1 shared Alex Cebria-Xart· 1 shared Alexandra AvgustinovaiD· 1 shared Alicia Castaneda· 1 shared Amir Z. MuniriD· 1 shared Andrea Onieva· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

Adria Caballe

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MET inhibition exploits conserved regulatory dependencies in malignant rhabdoid tumors

Ines Sentis, Alex Cebria-Xart, Quirze Rovira, Elisa Melon-Ardanaz, Carlos J. Rodriguez Hernandez, Samuel Morabito, Adria Caballe, Irene Ruano, Andrea Urpi, Maria Caballero, Sara Garcia Palomino, Eva Rodriguez, Davide Maspero, Ginevra Caratu, Andrea Onieva, , Alexandra Avgustinova

This study identifies MET signaling as a targetable vulnerability in rhabdoid tumors through integrated single-cell multi-omic and spatial transcriptomics analyses.

bioRxiv · Nov 4, 2026 · PreprintCancer Biology

A CD4-CD8 T-cell circuit converts cardiac inflammation into tissue injury

Amir Z. Munir, Alan Gutierrez, C M Krawiec, Riad Ghandour, Richard A. Baylis, Anya C. Shyani, Eva Rodriguez, Enrique Ortega‐Sollero, Aishwarya Nene, Javid J. Moslehi

This study shows that CD4-CD8 T-cell interactions are necessary for cardiac injury, as perforin-dependent cytotoxicity drives tissue damage despite ongoing inflammation.

bioRxiv · Sep 24, 2026 · PreprintImmunology

Diverse transcriptomic and mutational patterns but limited functional pathway alterations in patient-derived SS cells

Fernando Gallardo, Evelyn Andrades, Arnau Iglesias, María Maqueda, Teresa Lobo‐Jarne, Jessica González, Joan Bertran, David Conde‐Estévez, Eva Rodriguez, Beatríz Bellosillo, Ramon Maria Pujol, Anna Bigas, Lluı́s Espinosa

The paper analyzes patient-derived SS cells to identify transcriptomic, mutational patterns, and functional pathway alterations, finding limited pathway changes but potential for MALT1 inhibition in therapy.

bioRxiv · Mar 7, 2025 · PreprintCancer Biology