Author

F. Capon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

C. Smith· 1 shared D. A. Burden· 1 shared F. D. Ciccarelli· 1 shared G. Lombardi· 1 shared H. J. Lachmann· 1 shared H. S. Young· 1 shared I. M. Carr· 1 shared J. N. Barker· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

C. Smith

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1 paper

Recent·Most cited

Interleukin-36 promotes systemic Type-I IFN responses in severe psoriasis.

M. Catapano, M. Vergnano, M. Romano, S. K. Mahil, S.-E. Choon, D. A. Burden, H. S. Young, I. M. Carr, H. J. Lachmann, G. Lombardi, C. Smith, F. D. Ciccarelli, J. N. Barker, F. Capon

The paper identifies an IL-36/TLR-9/Type-I IFN axis that drives systemic inflammation in psoriasis, offering a potential therapeutic target.

bioRxiv · Dec 14, 2018 · PreprintImmunology