Author

I. Lin

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Wei· 1 shared B. Russell· 1 shared R. A. Weksberg· 1 shared V. A. Arboleda· 1 shared Z. Awamleh· 1 shared

Activity by Year

2022-2022 · peak 1/yr

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5 top collaborators · 10 mutual co-author links

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1 shared paperCancer Biology

A. Wei

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1 paper

Recent·Most cited

ASXL1 mutations that cause Bohring Opitz Syndrome (BOS) or acute myeloid leukemia share epigenomic and transcriptomic signatures

I. Lin, Z. Awamleh, A. Wei, B. Russell, R. A. Weksberg, V. A. Arboleda

This study compares epigenomic and transcriptomic changes in Bohring-Opitz Syndrome and AML with ASXL1 mutations, finding shared dysregulation and potential common therapeutic targets.

bioRxiv · Dec 15, 2022 · PreprintCancer Biology