Author

J. Epperlein

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

C. Kutter· 1 shared C. Sommerauer· 1 shared E. K. Brinkman· 1 shared K. Geng· 1 shared L. G. Merino· 1 shared R. G. Veiga· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 15 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

C. Kutter

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

CRISPR-Cas9-mediated genome engineering exaggerates genomic deletion at 10q23.31 including the PTEN gene locus mimicking cancer profiles

K. Geng, L. G. Merino, R. G. Veiga, C. Sommerauer, J. Epperlein, E. K. Brinkman, C. Kutter

This study identifies an unintended large genomic deletion at 10q23.31 in CRISPR-Cas9-modified HAP1 cells, which mimics cancer profiles and affects gene expression and histone modifications.

bioRxiv · Apr 5, 2023Cancer Biology