Author

J. M. Cunningham

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Ecology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:Arabidopsis· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Belemvire· 1 shared A. G. Orellana Herrera· 1 shared A. Mendoza· 1 shared B. Jiang· 1 shared C. Duque· 1 shared C. M. Browne· 1 shared C. Torres Gutierrez· 1 shared C. Yurrita· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperEcology

A. Belemvire

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The need to harmonize insecticide resistance testing: methodology, intensity concentrations and molecular mechanisms evaluated in Aedes aegypti populations in Central America and Hispaniola

S. Ledoux, C. Torres Gutierrez, N. F. Lobo, E. M. Murillo, S. Perez, R. Guerra, S. Chanquin Avendano, A. G. Orellana Herrera, A. Mendoza, D. Escobar, G. Guzman Contreras, M. Rivera, G. Ventura, R. Anagonou, E. Pierre-Louis, , N. Grisales

This study evaluates insecticide resistance in Aedes aegypti across Central America and Hispaniola, finding widespread resistance to pyrethroids and organophosphates, and identifies the V1016I kdr mutation as a key mechanism.

bioRxiv · Feb 25, 2020 · PreprintEcology

Targeting the PI5P4K lipid kinase family in cancer using novel covalent inhibitors

S. C. Sivakumaren, H. Shim, T. Zhang, F. M. Ferguson, M. R. Lundquist, C. M. Browne, H.-S. Seo, M. N. Paddock, T. D. Manz, B. Jiang, M.-F. Hao, P. Krishnan, D. G. Wang, T. J. Yang, N. P. Kwiatkowski, , N. S. Gray

This study identifies THZ-P1-2, a covalent pan-PI5P4K inhibitor, showing efficacy in AML/ALL cell lines and autophagy disruption, supporting PI5P4Ks as therapeutic targets in cancer.

bioRxiv · Oct 25, 2019Cancer Biology