Author

Matthew Unger

University of Massachusetts Chan Medical SchoolPrimary ORCID profile (0009-0002-6166-5826 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Matthew UngeriD1 paper · University of California, Santa Barbara

Biology Disciplines

Click to filter author papers

Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Cathleen M. LutziD· 1 shared Daryl A. BoscoiD· 1 shared Debra Cameron· 1 shared Del Hayden GaddiD· 1 shared Dorothy P. SchaferiD· 1 shared Elaine T. LimiD· 1 shared Francesca MassiiD· 1 shared Jeffrey A. NickersoniD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Cathleen M. LutzORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Expression of ALS-PFN1 impairs vesicular degradation in iPSC-derived microglia

Salome Funes, Jonathan Jung, Del Hayden Gadd, Michelle Mosqueda, Jianjun Zhong, Shankaracharya, Matthew Unger, Karly M. Stallworth, Debra Cameron, Melissa S. Rotunno, Pepper Dawes, Megan Fowler, Pamela J. Keagle, Justin A. McDonough, Sivakumar Boopathy, , Daryl A. Bosco

This study shows that ALS-linked PFN1 mutations impair vesicular degradation in iPSC-derived microglia by disrupting autophagy, which can be mitigated by Rapamycin.

Nature Communications · Mar 20, 2024Peer-reviewedNeuroscience