Author

Ravi Agarwal

Salk Institute for Biological Studies

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Assay:metabolomics· 1

Frequent Co-Authors

Fred H. GageiD· 2 shared Jeffrey R. JonesiD· 2 shared Michael S. CuocoiD· 2 shared Thaís S. SabedotiD· 2 shared Alison Jane YarnalliD· 1 shared Alwena H. MorganiD· 1 shared Benjamin J. JenkinsiD· 1 shared Bethan R DavidiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Fred H. GageORCID

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Author profile →

2 papers

Recent·Most cited

An age-preserving neuronal iSpheroid platform for modeling human aging and Alzheimer disease

Samhan Alsolami, Meiyan Wang, Thaís S. Sabedot, Upendra Singh, Brian Kirk, Lisa E. Mitchell, Melissa Hernandez, Nicholas J. Skvir, Chynna Bowman, Kotaro Oiwa, Ravi Agarwal, Marjan Mahmoudi, Michael S. Cuoco, Elise MacDonald, Samuel Cranston, , Fred H. Gage

This paper introduces an age-preserving neuronal iSpheroid platform that models human aging and Alzheimer's disease, revealing disease-associated metabolic changes and potential therapeutic targets.

bioRxiv · Oct 1, 2026 · PreprintNeuroscience

Aberrant CD4+ T cell refeeding response impairs neuro-immune crosstalk in Parkinson's disease

Karl Austin‐Muttitt, Bethan R David, Martina Sassi, Ravi Agarwal, Thaís S. Sabedot, Jeffrey R. Jones, Michael S. Cuoco, Benjamin J. Jenkins, Luke D. Roberts, Leo Harris, Natalia Kieronczyk, Gang Han, Matthew D. Hitchings, Paul C. Donaghy, Alison Jane Yarnall, , Jeffrey S. Davies

The paper shows that PD patients have altered CD4+ T cell responses to fasting/refeeding, leading to impaired neuro-immune signaling through a dysfunctional metabolic program in neurons.

bioRxiv · May 11, 2026 · PreprintNeuroscience