Author

S. Karahuseyinoglu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Assay:ChIP-seq· 1 Assay:clinical cohort / trial· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Cingoz· 1 shared A. Demiray· 1 shared A. Kayabolen· 1 shared A. P. Cribbs· 1 shared B. Isik· 1 shared B. K. Kilicoglu· 1 shared C. Vatansever· 1 shared D. P. Cahill· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Cingoz

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Orthogonal targeting of KDM6A/B and HDACs mediates potent therapeutic effects in IDH1-mutant glioma

A. Kayabolen, E. Yilmaz, G. N. Sahin, F. Seker-Polat, A. Cingoz, B. Isik, S. Acar, S. Karahuseyinoglu, H. Wakimoto, D. P. Cahill, I. Solaroglu, A. P. Cribbs, U. Oppermann, T. Bagci-Onder

This study identifies GSK-J4 and Belinostat as effective therapies for IDH1-mutant glioma by targeting KDM6A/B and HDACs, showing synergistic effects in vitro and in vivo.

bioRxiv · Nov 26, 2020 · PreprintCancer Biology

The role of iron uptake systems in the pathogenesis of colistin-resistant hypervirulent K. pneumoniae infections

O. Dogan, C. Vatansever, N. Atac, O. Albayrak, S. Karahuseyinoglu, O. E. Sahin, B. K. Kilicoglu, A. Demiray, O. Ergonul, M. Gönen, F. Can

This study identifies iron uptake systems as critical for survival of hypervirulent colistin-resistant K. pneumoniae against neutrophils and suggests targeting these systems as a therapeutic strategy.

bioRxiv · Jun 21, 2019 · PreprintMicrobiology