Author

Wu Wei

Stanford University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioinformatics· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:benchmarking· 2 Assay:spatial transcriptomics· 2 Approach:benchmark study· 1 Assay:bulk RNA-seq· 1 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

Chao XueiD· 1 shared Chenchen ZhuiD· 1 shared Chengming ZhangiD· 1 shared Chengyu LiiD· 1 shared Cirong LiuiD· 1 shared Han SuniD· 1 shared Hong ZengiD· 1 shared Jiang CheniD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperBioinformatics

Chao XueORCID

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3 papers

Recent·Most cited

SMECT: a framework for benchmarking post-GWAS methods for spatial mapping of cells associated with human complex traits

Meng Liu, Chao Xue, Y H Luo, Wenjie Peng, Lihang Ye, Liubin Zhang, Wu Wei, Miaoxin Li

SMECT benchmarks post-GWAS methods for spatial mapping of cells associated with human complex traits, revealing trade-offs between sensitivity and specificity across methods.

bioRxiv · Feb 16, 2026 · PreprintBioinformatics

BrainBeacon: A Cross-Species Foundation Model for Single-cell Resolved Brain Spatial Transcriptomics

Chengming Zhang, Yiwen Yang, Yifeng Jiao, Qianqian Yang, Xin Guo, Jing Xu, Jiyang Li, Yuanyuan Zhou, Zhen Liu, Yushuai Wu, Yu Zhang, Jiang Chen, Jianyun Ma, Yuxuan Liu, Yao Zhang, , Wu Wei

BrainBeacon is a cross-species foundation model for whole-brain spatial transcriptomics that enables cross-species alignment and uncovers spatial regulatory mechanisms of aging.

bioRxiv · Jul 8, 2025 · PreprintBioinformatics

Transcript fusion between CDKN1A and RAB44 caused by exon skipping like mechanism due to disruption of a splice site

Han Sun, Michelle Nguyen, William F. Mueller, Zhuanfen Cheng, Hong Zeng, Chenchen Zhu, Jingyan Wu, Kevin R. Roy, Petra Jakob, Raeka S. Aiyar, Wu Wei, Lars Michael Steinmetz

The paper identifies a recurrent tumor-specific transcript fusion between CDKN1A and RAB44 caused by splice-site mutations and shows its correlation with TP53 inactivation and CDK activation.

bioRxiv · Feb 22, 2019 · PreprintGenetics