Author

A. Snoberger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Approach:microscopy & imaging analysis· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1 Organism:yeast· 1

Frequent Co-Authors

E. M. Ostap· 2 shared B. Barua· 1 shared D. A. Winkelmann· 1 shared D. G. Drubin· 1 shared D. Safer· 1 shared E. Forgacs· 1 shared H. Shuman· 1 shared J. L. Atherton· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

E. M. Ostap

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Endocytic myosin-1 is a force-insensitive, power-generating motor

R. T. Pedersen, A. Snoberger, S. Pyrpassopoulos, D. Safer, D. G. Drubin, E. M. Ostap

The paper measures the force-sensitivity of yeast endocytic myosin Myo5 and finds it generates power rather than acting as a force-sensitive anchor during endocytosis.

bioRxiv · Mar 21, 2023 · PreprintCell Biology

Human hypertrophic cardiomyopathy mutation R712L suppresses the working stroke of cardiac myosin and can be rescued by omecamtiv mecarbil

A. Snoberger, B. Barua, J. L. Atherton, H. Shuman, E. Forgacs, Y. E. Goldman, D. A. Winkelmann, E. M. Ostap

This study shows that the HCM mutation R712L in β-cardiac myosin reduces the working stroke, which is reversed by the drug omecamtiv mecarbil.

bioRxiv · Oct 2, 2020Biophysics