Author

A. Turdo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Fasciani· 2 shared a. Zippo· 2 shared M. Todaro· 2 shared V. Poli· 2 shared A. Cherubini· 1 shared A. Chinnici· 1 shared A. Miluzio· 1 shared E. Lipari· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. Fasciani

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

ANP32E drives vulnerability to ATR inhibitors by inducing R-loops-dependent Transcription Replication Conflicts in Triple Negative Breast Cancer

S. Lago, V. Poli, L. Fol, M. Botteon, A. Fasciani, A. Turdo, M. Gaggianesi, M. Todaro, Y. Ciani, G. D Amato, F. Demichelis, a. Zippo

The paper identifies ANP32E as a driver of vulnerability to ATR inhibitors in triple-negative breast cancer by promoting R-loop-dependent transcription replication conflicts and genomic instability.

bioRxiv · Mar 25, 2024Cancer Biology

MYC-driven epigenetic reprogramming favors the onset of tumorigensis by inducing a stem cell-like state

V. Poli, L. Fagnocchi, A. Fasciani, A. Cherubini, S. Mazzoleni, S. Ferrillo, A. Miluzio, G. Gaudioso, V. Vaira, A. Turdo, M. Giaggianesi, A. Chinnici, E. Lipari, S. Bicciato, S. Bosari, , A. Zippo

This study shows MYC induces an epigenetic reprogramming in mammary epithelial cells that promotes a stem cell-like state and tumor initiation in breast cancer.

bioRxiv · Dec 29, 2017Cancer Biology