Author

B. J. Drapkin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. F. Farago· 1 shared B. Freitas· 1 shared C. B. Meador· 1 shared C. Gilbreath· 1 shared C. L. Christensen· 1 shared C. Rao· 1 shared D. Chatterjee· 1 shared D. H. Huisman· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. F. Farago

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2 papers

Recent·Most cited

KSR1 mediates small-cell lung carcinoma tumor initiation and cisplatin resistance

D. Chatterjee, R. A. Svoboda, D. H. Huisman, B. J. Drapkin, H. M. Vieira, C. Rao, J. W. Askew, K. W. Fisher, R. E. Lewis

This study identifies KSR1 as a critical regulator of SCLC tumor initiation and cisplatin resistance, showing that its disruption enhances cisplatin toxicity and reduces tumor-initiating cells.

bioRxiv · Feb 23, 2024Cancer Biology

Acquired Cross-resistance in Small Cell Lung Cancer due to Extrachromosomal DNA Amplification of MYC paralogs

S. Pal Choudhuri, L. Girard, J. Y. S. Lim, J. F. Wise, B. Freitas, D. Yang, E. Wong, S. Hamilton, V. Chien, S. Phat, C. Gilbreath, J. Zhong, D. T. Myers, C. L. Christensen, M. Stanzione, , B. J. Drapkin

This paper identifies extrachromosomal DNA amplification of MYC paralogs as a recurrent driver of acquired cross-resistance in small cell lung cancer using patient-derived xenograft models.

bioRxiv · Jun 23, 2023 · PreprintCancer Biology