Author

D. H. Huisman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 3 Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

D. Chatterjee· 3 shared H. M. Vieira· 3 shared K. W. Fisher· 3 shared R. A. Svoboda· 3 shared R. E. Lewis· 3 shared C. Rao· 2 shared J. W. Askew· 2 shared A. S. Ireland· 1 shared

Activity by Year

2022-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersCancer Biology

D. Chatterjee

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cancer BiologybioRxivDec 6, 2022

SOS1 and KSR1 modulate MEK inhibitor responsiveness to target resistant cell populations based on PI3K and KRAS mutation status

B. Daley, H. M. Vieira, C. Rao…·R. L. Kortum

This study shows that targeting SOS1 or KSR1 enhances MEK inhibitor efficacy and prevents resistance in KRAS-mutated cancers, depending on specific KRAS and co-mutation status.

DOI 10.1073/pnas.2313137120

humanbulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivPreprintMay 16, 2022

Post-transcriptional control of SRSF9 promotes the epithelial-to-mesenchymal transition (EMT) in colorectal cancer cells

C. Rao, R. A. Svoboda, S. Southekal…·R. E. Lewis

The paper shows that KSR1-dependent signaling in CRC cells promotes SRSF9-mediated splicing of EPSTI1 mRNA, which drives EMT and metastasis.

DOI 10.1101/2022.05.16.492181

humanmultiome

𝕏 PostBluesky

Cancer BiologybioRxivFeb 11, 2022

KSR2 promotes self-renewal and clonogenicity of small-cell lung carcinoma

D. H. Huisman, D. Chatterjee, R. A. Svoboda…·R. E. Lewis

This study shows KSR2 promotes self-renewal and clonogenicity in ASCL1-subtype SCLC by enhancing ERK signaling and tumor initiating capacity.

DOI 10.1158/1541-7786.mcr-24-0546

humanbulk RNA-seq

𝕏 PostBluesky