Author

Benedict E Ridore

Washington University in St. Louis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Abraham Ogbaslase· 1 shared Alexandria J. SturtziD· 1 shared Angela S. Archambault· 1 shared Gregory F. WuiD· 1 shared Joshua Amosu· 1 shared Kaitlyn Ying· 1 shared Kia M. BarclayiD· 1 shared Peggy L. KendalliD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Abraham Ogbaslase

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1 paper

Recent·Most cited

Distinct contribution of autoreactive B cell Bruton's tyrosine kinase signaling to neuroinflammation

Abraham Ogbaslase, Angela S. Archambault, Kia M. Barclay, Benedict E Ridore, Joshua Amosu, Kaitlyn Ying, Sravanthi Bandla, Alexandria J. Sturtz, Qingyun Li, Peggy L. Kendall, Gregory F. Wu

This study shows that deleting BTK in B cells reduces EAE severity by impairing autoreactive B cell functions, supporting BTK as a therapeutic target in MS.

bioRxiv · Apr 20, 2026 · PreprintImmunology