Author

D. Bulavin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Emelyanov· 1 shared B. B. Grigorash· 1 shared B. Kanzler· 1 shared B. Keyes· 1 shared C. Garrido· 1 shared C. Molina· 1 shared C. Spiegelhalter· 1 shared D. Sampaio Goncalves· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. Emelyanov

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Fragmentation through cell-cell adhesion maintains senescent cell viability but promotes debris deposition

M. Durik, D. Sampaio Goncalves, T. Knauer Meyer, C. Spiegelhalter, N. Messaddeq, M. T. Seehawer, D. Bulavin, L. Zender, B. Keyes

This study identifies senescent-cell adhesion fragments (SCAFs) as a mechanism for senescent cell survival, but also for the release of damaged contents that may contribute to disease.

bioRxiv · Jan 11, 2023 · PreprintCell Biology

p16High senescence restricts totipotent potential during somatic cell reprogramming

B. B. Grigorash, D. van Essen, L. Grosse, A. Emelyanov, B. Kanzler, C. Molina, E. Lopez, O. N. Demidov, C. Garrido, S. Saccani, D. Bulavin

This study shows that removing p16High senescent cells enhances 4F-induced reprogramming of somatic cells into totipotent-like stem cells by modulating NNMT and SAM levels.

bioRxiv · Aug 24, 2022 · PreprintCell Biology