Author

B. Kanzler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

A. Emelyanov· 1 shared B. B. Grigorash· 1 shared C. Garrido· 1 shared C. Molina· 1 shared D. Bulavin· 1 shared D. van Essen· 1 shared E. Lopez· 1 shared L. Grosse· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. Emelyanov

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1 paper

Recent·Most cited

p16High senescence restricts totipotent potential during somatic cell reprogramming

B. B. Grigorash, D. van Essen, L. Grosse, A. Emelyanov, B. Kanzler, C. Molina, E. Lopez, O. N. Demidov, C. Garrido, S. Saccani, D. Bulavin

This study shows that removing p16High senescent cells enhances 4F-induced reprogramming of somatic cells into totipotent-like stem cells by modulating NNMT and SAM levels.

bioRxiv · Aug 24, 2022 · PreprintCell Biology