Author

D. Hohl

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

M. Huber· 2 shared A. Smahi· 1 shared B. Wollnik· 1 shared C. Pich· 1 shared C. Pich Bavastro· 1 shared D. Bachmann· 1 shared E. Chiticariu· 1 shared E. Papanastasi· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

M. Huber

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Author profile →

2 papers

Recent·Most cited

TRAIP regulates DNA double-strand break-induced ATM activation

T. Gleich, M. Quadroni, G. Yigit, B. Wollnik, M. Huber, C. Pich Bavastro, D. Hohl

This study identifies TRAIP as a novel regulator of ATM activation in response to DNA double-strand breaks, showing its role in maintaining ATM downstream targets and DNA repair.

bioRxiv · Oct 11, 2021 · PreprintCell Biology

ARP-T1 is a ciliogenesis protein associated with a novel ciliopathy in inherited basal cell cancer, Bazex-Dupre-Christol Syndrome

H.-S. Park, E. Papanastasi, G. Blanchard, E. Chiticariu, D. Bachmann, M. Plomann, F. Morice-Picard, P. Vabres, A. Smahi, M. Huber, C. Pich, D. Hohl

This study identifies ARP-T1 as a ciliogenesis protein whose mutations cause Bazex-Dupre-Christol Syndrome, linking it to ciliopathy and skin cancer.

bioRxiv · Dec 16, 2019 · PreprintCancer Biology