Author

D. M. Jensen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:human· 1

Frequent Co-Authors

B. C. Hofeld· 1 shared B. M. Murphy· 1 shared C. E. Burd· 1 shared C. J. Burd· 1 shared D. Z. Trykall· 1 shared F. C. Peterson· 1 shared J. Hughes· 1 shared J. M. Egner· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

B. C. Hofeld

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The OSUMMER lines: a series of ultraviolet-accelerated NRAS-mutant mouse melanoma cell lines syngeneic to C57BL/6

B. M. Murphy, D. M. Jensen, T. E. Arnold, R. Aguilar Valenzuela, J. Hughes, V. Posada, K. T. Nguyen, V. T. Chu, K. Y. Tsai, C. J. Burd, C. E. Burd

The paper introduces a series of NRAS-mutant melanoma cell lines derived from UV-exposed mice to model human melanoma resistance and therapy responses.

Pigment Cell & Melanoma Research · Sep 1, 2023Peer-reviewedCancer Biology

A novel Fis1 inhibiting peptide reverses diabetic endothelial dysfunction in human resistance arteries

K. A. Nolden, M. Kakarla, J. M. Egner, J. Wang, M. C. Harwig, V. K. Puppala, B. C. Hofeld, L. A. Arnold, D. Z. Trykall, F. C. Peterson, M. L. Roberts, D. M. Jensen, R. B. Hill, M. E. Widlansky

This study shows that inhibiting Fis1 improves endothelial function in diabetic vessels by enhancing NO availability and reducing mitochondrial ROS.

bioRxiv · Nov 16, 2020 · PreprintPhysiology