Author

D. Trouche

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 3 Approach:experimental assay· 3 Organism:human· 3 Assay:ChIP-seq· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Constantinou· 1 shared C. Chailleux· 1 shared C. Jacques· 1 shared C. Leoni· 1 shared C. Ribeyre· 1 shared C. Rives· 1 shared F. Escaffit· 1 shared J. Fages· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCancer Biology

A. Constantinou

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Control of intestinal stemness and cell lineage by histone variant H2A.Z isoforms

J. Rispal, C. Rives, V. Jouffret, C. Leoni, L. Dubois, M. Chevillard-Briet, D. Trouche, F. Escaffit

This study investigates the role of H2A.Z isoforms in intestinal homeostasis, showing they regulate stemness, lineage differentiation, and enterocyte differentiation with opposing effects.

Molecular and Cellular Biology · Aug 18, 2024Peer-reviewedPhysiology

KDM5 histone-demethylases contribute to replication stress response and tolerance.

M. Vandromme, S. Gaillard, V. Charasson, C. Ribeyre, K. Salifou, M.-J. Pillaire, J.-S. Hoffmann, A. Constantinou, D. Trouche

This study shows KDM5A and KDM5B contribute to replication stress response and tolerance through mechanisms independent of their demethylase activity.

bioRxiv · Dec 16, 2019 · PreprintCancer Biology

JMJD6 participates in the maintenance of ribosomal DNA integrity in response to DNA damage

J. Fages, C. Chailleux, J. Humbert, S.-M. Jang, J. Loehr, J.-P. Lambert, C. Jacques, D. Trouche, y. canitrot

This study identifies JMJD6 as essential for rDNA stability during DNA damage by facilitating nucleolar cap formation and NBS1 recruitment, independent of rDNA transcription inhibition.

bioRxiv · Nov 6, 2019Cell Biology