Author

E. Knapp

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 3 Biochemistry· 1 Bioengineering· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 5 Organism:mouse· 5 Research Focus:development & morphogenesis· 4 Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:clustering· 1

Frequent Co-Authors

A. Loiselle· 2 shared V. Studentsova· 2 shared A. E. Loiselle· 1 shared A. J. Berger· 1 shared A. Korcari· 1 shared A. Paine· 1 shared B. H. Shares· 1 shared C. Massie· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 9 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

A. Loiselle

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Improved prediction of femoral fracture toughness in mice by combining standard medical imaging with Raman spectroscopy

C. Massie, E. Knapp, K. Chen, A. J. Berger, H. A. Awad

This study combines DXA and Raman spectroscopy to predict femoral fracture toughness in mice, showing improved accuracy with integrated data.

bioRxiv · Nov 20, 2020Bioengineering

Scleraxis-Lineage Cell Depletion Improves Tendon Healing and Disrupts Adult Tendon Homeostasis

K. T. Best, A. Korcari, K. E. Mora, E. Knapp, M. R. Buckley, A. E. Loiselle

This study depletes Scleraxis-lineage cells in adult tendons and finds improved biomechanical properties and altered collagen structure, suggesting a role in homeostasis rather than repair.

bioRxiv · Feb 1, 2020Cell Biology

Inhibition of the mitochondrial permeability transition exerts sex-specific stimulatory effect on fracture repair

B. H. Shares, T.-J. Sheu, K. Schilling, R. Sautchuk, A. Paine, G. Beutner, L. C. Shum, C. O. Smith, E. Knapp, E. B. Brown, H. Awad, R. A. Eliseev

The paper shows that inhibiting CypD improves fracture repair in male mice but not females, with effects linked to mitochondrial function and β-catenin activity.

bioRxiv · Sep 18, 2019 · PreprintBiochemistry

Cell non-autonomous functions of S100a4 drive fibrotic tendon healing

J. Ackerman, V. Studentsova, K. Best, E. Knapp, A. Loiselle

This study shows that inhibiting S100a4 improves tendon healing by reducing scar formation and enhancing mechanical properties through cell non-autonomous mechanisms.

bioRxiv · Jan 9, 2019Cell Biology

Insulin Receptor Deletion in S100a4-lineage cells accelerates age-related bone loss

V. Studentsova, E. Knapp, A. Loiselle

This study deletes the insulin receptor in S100a4-lineage cells in mice and finds accelerated age-related bone loss in older adults.

bioRxiv · Oct 16, 2018Cell Biology