Author

E. McNally

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

A. M. Gacita· 1 shared C. D. Morsink· 1 shared H. Zahr· 1 shared I. J. Benjamin· 1 shared J. Lammerding· 1 shared L. E. Johnson· 1 shared L. L. Wallrath· 1 shared M. Wallace· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. M. Gacita

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1 paper

Recent·Most cited

Nuclear damage in LMNA mutant iPSC-derived cardiomyocytes is associated with impaired lamin localization to the nuclear envelope

M. Wallace, H. Zahr, S. Perati, C. D. Morsink, L. E. Johnson, A. M. Gacita, S. Lai, L. L. Wallrath, I. J. Benjamin, E. McNally, T. J. Kirby, J. Lammerding

This study shows that LMNA mutations in iPSC-derived cardiomyocytes cause nuclear shape changes, reduced stiffness, and lamin mislocalization, linking nuclear abnormalities to LMNA-DCM pathogenesis.

bioRxiv · Oct 30, 2021 · PreprintCell Biology