Author

E. W. Schmid

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. S. Krall· 1 shared A. Thambundit· 1 shared C. A. Lyssiotis· 1 shared C. J. Halbrook· 1 shared D. B. Shackelford· 1 shared F. Surjono· 1 shared H. R. Christofk· 1 shared J. C. Walter· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. S. Krall

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2 papers

Recent·Most cited

STK19 positions TFIIH for cell-free transcription-coupled DNA repair

T. E. T. Mevissen, M. Kuemmecke, E. W. Schmid, L. Farnung, J. C. Walter

The paper investigates the role of STK19 in transcription-coupled DNA repair by showing it positions TFIIH for repair in a cell-free system.

bioRxiv · Jul 22, 2024Biochemistry

Asparagine signals mitochondrial respiration and can be targeted to impair tumour growth

A. S. Krall, P. J. Mullen, F. Surjono, M. Momcilovic, E. W. Schmid, C. J. Halbrook, A. Thambundit, S. D. Mittelman, C. A. Lyssiotis, D. B. Shackelford, S. R. Knott, H. R. Christofk

This study shows that asparagine, derived from mitochondrial respiration, supports mTORC1 activity and tumor growth, and that targeting asparagine synthesis or consumption can impair tumor growth.

bioRxiv · Mar 17, 2020 · PreprintCancer Biology