Author

E. Ziska

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1

Frequent Co-Authors

M. J. Taylor· 3 shared R. Deliz-Aguirre· 3 shared F. Cao· 2 shared F. H. U. Gerpott· 2 shared D. K. Gola· 1 shared M. Bilay· 1 shared M. Chupanova· 1 shared N. Auevechanichkul· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersImmunology

M. J. Taylor

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3 papers

Recent·Most cited

IRAK4 autophosphorylation controls inflammatory signaling by activating IRAK oligomerization

N. Srikanth, R. Deliz-Aguirre, D. K. Gola, M. Bilay, E. Ziska, M. J. Taylor

This study shows IRAK4 auto-phosphorylation activates IRAK oligomerization, controlling inflammatory signaling by promoting DD domain assembly and downstream IL-1 signaling.

bioRxiv · Dec 21, 2023 · PreprintImmunology

Myddosome clustering in IL-1 receptor signaling regulates the formation of an NF-kB activating signalosome

F. Cao, R. Deliz-Aguirre, F. H. U. Gerpott, E. Ziska, M. J. Taylor

The paper shows that Myddosome clustering in IL-1R signaling regulates NF-kB activation by forming a signalosome that enhances TRAF6 and LUBAC recruitment.

bioRxiv · Jan 6, 2023Cell Biology

On demand MyD88 oligomerization is controlled by IRAK4 during Myddosome signaling

R. Deliz-Aguirre, F. Cao, F. H. U. Gerpott, N. Auevechanichkul, M. Chupanova, Y. Mun, E. Ziska, M. J. Taylor

This study investigates how MyD88 oligomerization is regulated by IRAK4 during Myddosome signaling, revealing that oligomer size acts as a threshold for downstream signaling.

bioRxiv · Sep 3, 2020Immunology