Author

F. Rajabi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. A. Yurchenko· 1 shared A. Lehmann· 1 shared A. Sarasin· 1 shared C. F. M. Menck· 1 shared C. Nishigori· 1 shared C. Robert· 1 shared F. Morice-Picard· 1 shared H. Fassihi· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. A. Yurchenko

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

DNA damage-induced PARP/ALC1 activation leads to Epithelial-to-Mesenchymal transition stimulating homologous recombination.

F. Rajabi, R. Smith, W.-Y. Liu-Bordes, M. Schertzer, S. Huet, J.-A. Londono-Vallejo

This study shows DNA damage activates PARP and ALC1 to induce EMT, which enhances DNA repair via RAD51, and PARP inhibitors block this process.

bioRxiv · Jan 16, 2024 · PreprintCancer Biology

Analysis of Skin Cancers from Xeroderma Pigmentosum Patients Reveals Heterogeneous UV-Induced Mutational Profiles Shaped by DNA Repair

A. A. Yurchenko, F. Rajabi, T. Braz-Petta, H. Fassihi, A. Lehmann, C. Nishigori, I. Padioleau, K. Gunbin, L. Panunzi, F. Morice-Picard, P. Laplante, C. Robert, P. Kannouche, C. F. M. Menck, A. Sarasin, S. I. Nikolaev

The paper analyzes skin cancer genomes from Xeroderma Pigmentosum patients to show how NER activity shapes mutation rates and reveals POLH's role in bypassing UV-induced DNA lesions.

bioRxiv · Oct 14, 2022 · PreprintCancer Biology