Author

G. Blandano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Cossa· 1 shared A. Dalmasso· 1 shared A. Polazzi· 1 shared A. Tirelli· 1 shared C. Priami· 1 shared E. Gatti· 1 shared E. Migliaccio· 1 shared F. Ruscitto· 1 shared

Activity by Year

2022-2022 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Cossa

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1 paper

Recent·Most cited

Cancer BiologybioRxivJul 13, 2022

The breast cancer pro-metastatic phenotype requires concomitant hyper-activation of ECM remodeling and dsRNA-IFN1 signaling in rare clone cells

N. Roda, A. Cossa, R. Hillje…·P. G. Pelicci

This study identifies that the breast cancer pro-metastatic phenotype arises from rare clones with hyper-activation of ECM remodeling and dsRNA-IFN1 signaling, and shows these genes predict metastasis progression.

DOI 10.1158/0008-5472.can-22-2717

humanscRNA-seq