Author

C. Priami

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Cossa· 2 shared N. Roda· 2 shared P. G. Pelicci· 2 shared V. Gambino· 2 shared A. Dalmasso· 1 shared A. Polazzi· 1 shared A. Tirelli· 1 shared E. Gatti· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

A. Cossa

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

INDOCYANINE GREEN IMAGING BY VITAL TISSUE SLICES SCANNING ALLOWS FOR THE ISOLATION OF INTACT LIVER MICRO-METASTASIS FOR SINGLE-CELL ANALYSIS

N. Roda, M. Stendardo, V. Gambino, G. Persico, A. Cossa, S. Rodighiero, C. Priami, N. Kaludercic, P. G. Pelicci, M. Giorgio

This study uses ICG imaging to isolate liver micro-metastases for single-cell analysis, revealing gene expression patterns predictive of breast cancer prognosis and therapeutic targets.

bioRxiv · Jun 21, 2023 · PreprintCancer Biology

The breast cancer pro-metastatic phenotype requires concomitant hyper-activation of ECM remodeling and dsRNA-IFN1 signaling in rare clone cells

N. Roda, A. Cossa, R. Hillje, A. Tirelli, F. Ruscitto, S. Cheloni, C. Priami, A. Dalmasso, V. Gambino, G. Blandano, A. Polazzi, P. Falvo, E. Gatti, L. Mazzarella, L. Luzi, , P. G. Pelicci

This study identifies that the breast cancer pro-metastatic phenotype arises from rare clones with hyper-activation of ECM remodeling and dsRNA-IFN1 signaling, and shows these genes predict metastasis progression.

bioRxiv · Jul 13, 2022Cancer Biology