Author

G. Burke

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Evolutionary Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Organism:mouse· 1 Approach:statistical model· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. Bretaudeau· 1 shared A. C. Rowat· 1 shared A. Lorenzi· 1 shared A.-N. Volkoff· 1 shared B. J. Sharanowski· 1 shared B. L. Krog· 1 shared C. S. Stipp· 1 shared D. L. Moose· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperMicrobiology

A. Bretaudeau

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Identification of a viral gene essential for the genome replication of a domesticated endogenous virus in ichneumonid parasitoid wasps

A. Lorenzi, F. Legeai, V. Jouan, P.-A. Girard, M. Strand, M. Ravallec, M. Eychenne, A. Bretaudeau, S. Robin, J. Rochefort, M. Villegas, G. Burke, R. Rebollo, N. Negre, A.-N. Volkoff

This study identifies a viral gene, U16, essential for genome replication of an ichnovirus in ichneumonid wasps, showing its role in DNA amplification and virion production.

PLOS Pathogens · Apr 25, 2024Peer-reviewedMicrobiology

Endogenization from diverse viral ancestors is common and widespread in parasitoid wasps

G. Burke, H. J. Hines, B. J. Sharanowski

This study identifies novel endogenous viral elements (EVEs) in parasitoid wasps, revealing widespread acquisition of diverse dsDNA viruses across multiple lineages.

bioRxiv · Jun 17, 2020Evolutionary Biology

Cancer cells resist mechanical destruction in the circulation via RhoA-myosin II axis

D. L. Moose, B. L. Krog, L. Zhao, T.-H. Kim, S. Williams-Perez, G. Burke, L. Rhodes, M. Vanneste, P. Breheny, M. Milhem, C. S. Stipp, A. C. Rowat, M. D. Henry

This study identifies the RhoA-myosin II axis as a mechanism enabling cancer cells to resist mechanical destruction by hemodynamic forces in circulation.

bioRxiv · Apr 6, 2019Cancer Biology