Author

G. Ricci

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Mangogna· 2 shared A. Martorana· 2 shared B. Belmonte· 2 shared F. Romano· 2 shared G. Zito· 2 shared A. Balduit· 1 shared A. Biran· 1 shared A. E. E. Verkennis· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersCell Biology

A. Mangogna

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PARP-1, EpCAM, and FRα as potential targets for intraoperative detection and delineation of endometriosis: a quantitative tissue expression analysis

B. Belmonte, G. Di Lorenzo, A. Mangogna, B. Bortot, G. Bertolazzi, S. Sammataro, S. Merighi, A. Martorana, G. Zito, F. Romano, A. Giorgiutti, C. Bottin, F. Zanconati, A. Romano, G. Ricci, S. Biffi

This study evaluates PARP-1, EpCAM, and FR as potential biomarkers for intraoperative detection of endometriosis through gene expression and immunohistochemical analysis.

bioRxiv · Dec 19, 2023Cell Biology

CAF-1 deposits newly synthesized histones during DNA replication using distinct mechanisms on the leading and lagging strands

C. Rouillon, B. V. Eckhardt, L. Kollenstart, F. Gruss, A. E. E. Verkennis, I. Rondeel, P. H. L. Krijger, G. Ricci, A. Biran, T. van Laar, C. M. Delvaux de Fenffe, G. Luppens, P. Albanese, R. A. Scheltema, W. de Laat, , F. Mattiroli

This study shows CAF-1 deposits histones on both DNA strands during replication but uses different mechanisms on leading and lagging strands.

bioRxiv · Oct 14, 2022Biochemistry

Complement Component 3 expressed by the endometrial ectopic tissue is involved in the endometriotic lesion formation through mast cell activation

C. Agostinis, S. Zorzet, A. Balduit, G. Zito, A. Mangogna, P. Macor, F. Romano, M. Toffoli, B. Belmonte, A. Martorana, V. Borelli, G. Ricci, U. Kishore, R. Bulla

This study shows that complement component 3 from endometrial ectopic tissue promotes endometriotic lesion formation by activating mast cells in a murine model.

bioRxiv · Nov 19, 2020 · PreprintImmunology