Author

J. Berrigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

E. Muhlberger· 2 shared J. Olejnik· 2 shared S. Gummuluru· 2 shared A. Nisa· 1 shared C. Costello· 1 shared C. Xia· 1 shared E. L. Suder· 1 shared E. Suder· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

E. Muhlberger

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2 papers

Recent·Most cited

CD169-mediated restrictive SARS-CoV-2 infection of macrophages induces pro-inflammatory responses

S. Jalloh, J. Olejnik, J. Berrigan, A. Nisa, E. L. Suder, H. Akiyama, M. Lei, S. Tyagi, Y. Bushkin, E. Muhlberger, S. Gummuluru

This study shows CD169 enables ACE2-independent SARS-CoV-2 entry in macrophages, leading to restricted viral replication and pro-inflammatory cytokine production.

bioRxiv · Mar 29, 2022Immunology

CD209L/L-SIGN and CD209/DC-SIGN act as receptors for SARS-CoV-2

R. Amraei, M. Napoleon, W. Yin, J. Berrigan, E. Suder, G. Zhao, J. Olejnik, K. Chandler, C. Xia, S. Gummuluru, E. Muhlberger, V. Chitalia, C. Costello, N. Rahimi

This study identifies CD209L/L-SIGN and CD209/DC-SIGN as alternative receptors for SARS-CoV-2, capable of mediating virus entry into human cells, particularly in tissues with low ACE2 expression.

bioRxiv · Jun 22, 2020 · PreprintCell Biology