Author

J. Gustafsson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Assay:CITE-seq· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. Hagnestal· 1 shared A. Martner· 1 shared D. F. Becker· 1 shared F. B. Thoren· 1 shared F. Roshanzamir· 1 shared H. Komic· 1 shared J. L. Robinson· 1 shared J. Nielsen· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

A. Hagnestal

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2 papers

Recent·Most cited

Multiomic single-cell analysis identifies von Willebrand factor and TIM3-expressing BCR-ABL1+ CML stem cells

M. S. Nilsson, H. Komic, J. Gustafsson, Z. Sheybani, S. Paul, O. Rolfson, K. Hellstrand, L. Wennström, A. Martner, F. B. Thoren

This study identifies VWF and TIM3-expressing BCR-ABL1+ CML stem cells using multiomic single-cell analysis, suggesting they are targets for sustained LSC elimination.

bioRxiv · Sep 14, 2023 · PreprintCancer Biology

Metabolic collaboration between cells in the tumor microenvironment has a negligible effect on tumor growth

J. Gustafsson, F. Roshanzamir, A. Hagnestal, S. M. Patel, O. I. Daudu, D. F. Becker, J. L. Robinson, J. Nielsen

The paper models metabolic interactions in the tumor microenvironment and finds that enzymatic capacity limits, not collaboration, drive cancer cell metabolism and growth.

bioRxiv · Feb 8, 2022Cancer Biology