Author

J. Levin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Almaguer· 1 shared A. Borchardt· 1 shared A. Elliott· 1 shared A. Kopczak· 1 shared A. Nalwoga· 1 shared A. Noncovich· 1 shared A. Zatcepin· 1 shared B. Foerstera· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperMicrobiology

A. Almaguer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

CD388: A universally protective Drug-Fc Conjugate that targets influenza virus neuraminidase

S. Dohrmann, J. Levin, J. N. Cole, A. Borchardt, K. Amundson, A. Almaguer, E. Abelovski, R. Grewal, D. Zuill, N. Dedeic, G. Hough, J. Fortier, J. Donatelli, T. Lam, Z.-Y. Chen, , L. W. Tari

This paper describes CD388, a drug-Fc conjugate that targets influenza virus neuraminidase, showing universal antiviral activity across multiple strains and demonstrating efficacy in mouse models.

bioRxiv · Jun 4, 2024 · PreprintMicrobiology

Multi-omics and 3D-imaging reveal bone heterogeneity and unique calvaria cells in neuroinflammation

Z. I. Kolabas, L. B. Kuemmerle, R. Perneczky, B. Foerstera, M. Buttner, O. S. Caliskan, M. Ali, Z. Rong, H. Mai, S. Hummel, L. M. Bartos, G. Biechele, A. Zatcepin, N. L. Albert, M. Unterrainer, , Brendel

The paper uses multi-omics and 3D-imaging to show that calvaria bone marrow has a distinct molecular profile and contributes to neuroinflammation.

bioRxiv · Dec 24, 2021 · PreprintCell Biology

Mycobacterium tuberculosis infection boosts B cell responses to unrelated pathogens

S. G. Kimuda, I. Andia-Biraro, I. Sebina, M. Egesa, A. Nalwoga, S. G. Smith, B. Ssentalo Bagaya, J. Levin, A. Elliott, J. G. Raynes, S. Cose

This study shows that Mycobacterium tuberculosis infection enhances B cell responses to unrelated pathogens, increasing antibody levels and plasmablast-to-memory B cell ratios.

bioRxiv · Jun 25, 2019Immunology