Author

K. C. OConnor

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:differential expression· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

K. B. Hoehn· 2 shared M. L. Fichtner· 2 shared R. J. Nowak· 2 shared R. Jiang· 2 shared C. Mandel-Brehm· 1 shared J. L. DeRisi· 1 shared M. C. Pham· 1 shared M. R. Wilson· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersImmunology

K. B. Hoehn

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2 papers

Recent·Most cited

Elevated N-glycosylation of immunoglobulin G variable regions in myasthenia gravis highlights a commonality across autoantibody-associated diseases

C. Mandel-Brehm, M. L. Fichtner, R. Jiang, V. J. Winton, S. E. Vazquez, M. C. Pham, K. B. Hoehn, N. L. Kelleher, R. J. Nowak, S. H. Kleinstein, M. R. Wilson, J. L. DeRisi, K. C. OConnor

This study identifies elevated N-glycosylation in IgG variable regions in myasthenia gravis patients and shows it is not required for autoantigen binding.

bioRxiv · Mar 6, 2021Immunology

Single-cell repertoire tracing identifies rituximab refractory B cells during myasthenia gravis relapses

R. Jiang, M. L. Fichtner, K. B. Hoehn, P. Stathopoulos, R. J. Nowak, S. Kleinstein, K. C. OConnor

The paper identifies persistent autoantibody-producing B cell clones in rituximab-treated myasthenia gravis patients during relapses, showing incomplete B cell depletion.

bioRxiv · Jan 15, 2020Immunology