Author

K. Lund

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

A. Corthay· 1 shared A. Tveita· 1 shared C. H. Stuelten· 1 shared C. Hammarstrom· 1 shared E. Dybing· 1 shared E. Hovig· 1 shared J. Waaler· 1 shared K.-M. Heintz· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Corthay

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A GPCR screening in human keratinocytes identifies that the metabolite receptor HCAR3 controls epithelial proliferation, migration, and cellular respiration.

M. P. Pedro, K. Lund, S. W. S. Kang, T. Chen, C. H. Stuelten, N. Porat-Shliom, R. Iglesias-Bartolome

This study identifies HCAR3 as a GPCR that regulates epithelial cell proliferation, migration, and respiration in human keratinocytes through altered metabolism and mitochondrial function.

bioRxiv · May 30, 2023Cell Biology

Tankyrase inhibition sensitizes melanoma to PD-1 immune checkpoint blockade in syngeneic mouse models

J. Waaler, L. Mygland, A. Tveita, M. F. Strand, N. T. Solberg, P. Angell Olsen, K. Lund, S. Alam Brinch, M. Lycke, E. Dybing, V. Nygaard, S. L. Boe, K.-M. Heintz, E. Hovig, C. Hammarstrom, , S. Krauss

This study shows that tankyrase inhibition makes melanoma more responsive to PD-1 checkpoint blockade in mouse models by reducing WNT/β-catenin and YAP signaling.

bioRxiv · Jan 22, 2019Cancer Biology