Author

L. Sole

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:host-pathogen & immune response· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Bigas· 3 shared L. Espinosa· 3 shared A. Villanueva· 2 shared D. Alvarez-Villanueva· 2 shared j. Bertran· 2 shared M. garrido· 2 shared M. Iglesias· 2 shared M. Martinez-Iniesta· 2 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

A. Bigas

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Separation-of-function mutants reveal the NF-κB-independent involvement of IκBα in the regulation of stem cell and oncogenic programs

D. Alvarez-Villanueva, L. Galan, j. Bertran, M. Floor, L. Sole, T. Lobo-Jarne, M. Maqueda, R. Rajbhandari, L. Marruecos, J. Villa, M. Bredel, A. Bigas, L. Espinosa

This paper identifies IκB's NF-κB-independent role in stem cell and oncogenic programs using separation-of-function mutants.

bioRxiv · Jun 21, 2023 · PreprintCell Biology

IKK1 kinase coordinates BRD4 and JAK/STAT signaling to subvert DNA damage-based anticancer therapy

L. Espinosa, I. Pecharroman Ruiz, L. Sole, D. Alvarez-Villanueva, T. Lobo, J. Alonso, j. Bertran, E. Sabido, E. Borras, V. Garcia-Hernandez, M. garrido, J. Seoane, A. Bigas, A. Villanueva, M. Martinez-Iniesta, , R. salazar

This study identifies IKK1's role in coordinating BRD4 and JAK/STAT signaling to resist DNA damage-based therapy in colorectal cancer.

bioRxiv · Jun 13, 2023 · PreprintCancer Biology

Chemotherapy induces a YAP1-dependent fetal conversion to human Colorectal Cancer cells that is predictive of poor patient outcome

L. Sole, T. Lobo-Jarne, A. Villanueva, A. Vert, Y. Guillen, I. Sangrador, A. Barbachano, J. Lop, M. Guix, M. Salido, B. Bellosillo, R. Garcia-Romero, M. Garrido, J. Gonzalez, M. Martinez-Iniesta, , L. Espinosa

The paper shows that sublethal chemotherapy induces a YAP1-dependent fetal phenotype in colorectal cancer cells, which predicts poor patient outcomes.

bioRxiv · Apr 8, 2021 · PreprintCancer Biology