Author

A. Barbachano

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

A. Munoz· 2 shared A. Arranz· 1 shared A. Bigas· 1 shared A. Chocarro-Calvo· 1 shared A. De la Vieja· 1 shared A. Vert· 1 shared A. Villanueva· 1 shared B. Bellosillo· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCancer Biology

A. Munoz

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Balance of AKT isoforms in intestinal barrier function. Implications for IBD therapy.

T. Garcia Prieto, A. Barbachano, N. G. Cuesta, D. SANCHEZ GONZALEZ, A. Arranz, M. Fresno

This study shows that balancing Akt1 and Akt2 isoforms regulates intestinal barrier function and inflammation, with Akt2 inhibition improving IBD symptoms in mice.

bioRxiv · Jul 4, 2023 · PreprintImmunology

Vitamin D induces SIRT1 activation through K610 deacetylation in colon cancer

J. M. Garcia-Martinez, A. Chocarro-Calvo, J. Martinez-Useros, M. J. Fernandez-Acenero, M. C. Fiuza, J. Caceres-Rentero, A. De la Vieja, A. Barbachano, A. Munoz, M. J. Larriba, C. Garcia Jimenez

This study shows vitamin D activates SIRT1 via K610 deacetylation in colon cancer, linking it to antiproliferative effects and suggesting therapeutic potential for VD-deficient patients.

bioRxiv · Feb 22, 2023Cancer Biology

Chemotherapy induces a YAP1-dependent fetal conversion to human Colorectal Cancer cells that is predictive of poor patient outcome

L. Sole, T. Lobo-Jarne, A. Villanueva, A. Vert, Y. Guillen, I. Sangrador, A. Barbachano, J. Lop, M. Guix, M. Salido, B. Bellosillo, R. Garcia-Romero, M. Garrido, J. Gonzalez, M. Martinez-Iniesta, , L. Espinosa

The paper shows that sublethal chemotherapy induces a YAP1-dependent fetal phenotype in colorectal cancer cells, which predicts poor patient outcomes.

bioRxiv · Apr 8, 2021 · PreprintCancer Biology